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We therefore postulated that IGF-1R mAbs could represent a viable therapeutic tool to target IGF-1 action, and potentially mimic the beneficial effects associated with diminished IGF-1 signaling observed in animal models
Gopi, S., & Balakrishnan, P
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non-GLP-1 RA medications and used propensity matching, Kaplan-Meier method, and Cox proportional hazards regression
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