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What made AOD 9604 compelling was the identification that a specific region of the hGH molecule, the C-terminal fragment, was responsible for this fat-burning effect, and that this region operated through a pathway separate from the growth hormone receptor

We recently reported that NNMTi treatment combined with a low-fat, or lean, diet (LD) returned systemic adiposity and liver pathologies of obese animals to the adiposity and liver pathology levels of LD controls

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Third, lipolysis becomes more efficient, allowing stored fat to be mobilized and used for energy

[DOI] [PubMed] [Google Scholar] Castelli MC, Wong D, Friedman K, Bhargava P, Riley GM