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The biological mechanism (localized melanin accumulation) is identical, which is why the corrective approach is also the same

Collagen-producing fibroblasts depend on adequate vascular supply to sustain their metabolic demand during active matrix synthesis, making angiogenesis a prerequisite for effective structural repair

312 Human neutrophils express FPR1 and FPR2, of which FPR1 binds fMLF specifically and rapidly, whereas FPR2 binds fMLF with low affinity

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