Note that 2-PAM has an affinity for the acetylcholine anionic binding site on cholinesterase (right middle panel), which could explain the oxime related worsening of cholinergic side effects when 2-PAM is administered prior to the onset of action of atropine in the setting of organophosphate poisoning, by transiently preventing nerve-agent free cholinesterase from interacting with acetylcholine
J Clin Invest 96(5):24212430 Santini F, Pinchera A, Ceccarini G et al (2001) Evidence for a role of the type III-iodothyronine deiodinase in the regulation of 3,5,3-triiodothyronine content in the human central nervous system
Combination procedures (face plus body, or any multi-site case): KLOW by default, since the inflammatory load wins
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[2] Is GHK-Cu the same as a copper peptide serum
