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This could imply that glucose is not efficiently converted to lactate in the astrocytes

Abstract We have previously proposed that the heterogeneous collapse of mitochondrial inner membrane potential ( m ) during ischemia and reperfusion contributes to arrhythmogenesis through the formation of metabolic sinks in the myocardium, wherein clusters of myocytes with uncoupled mitochondria and high K ATP current levels alter electrical propagation to promote reentry

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Biochem Mol Med