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GM-CSF, IL-6) [57, 91] might reprogram CAFs and immune cells towards an iron-donor phenotype (via transferrin or LCN2) to support tumor growth

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Moreover, knockdown of FAT1 significantly blocked the inhibitory effect of MV-carried OLFM4 on the Hippo signaling (Fig

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In addition, this result may exclude the possibility that (-oxo)(-hydroxo)-dicopper(II,III) acts as an active species for the hydroxylation, which was proposed from the simulation analysis [40], at least in our system

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