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Oxidative modifications to proteins, lipids, and/or DNA can alter the function of these molecules and may result in the activation of ER stress, mitochondrial dysfunction, autophagy, and apoptosis, processes that themselves also generate ROS [14]

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A novel RANKL-targeted flavonoid glycoside prevents osteoporosis through inhibiting NFATc1 and reactive oxygen species

ZFP36L2 is required for self-renewal of early burst-forming unit erythroid progenitors

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Since glutathione is used up more than its produced, injections are the best way to meet the demand