Critically, this redox adaptation is a key mechanism of resistance to radiotherapy and many chemotherapeutic agents, such as cisplatin and doxorubicin, which depend on generating oxidative damage to exert their cytotoxic effects [33]
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Human CHAC1 protein degrades glutathione, and mrna induction is regulated by the transcription factors ATF4 and ATF3 and a bipartite ATF/CRE regulatory element
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