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Concurrent activation of the ascorbateglutathione (AsA-GSH) cycle maintained redox equilibrium during early stress phases, though prolonged exposure imposed redox constraints

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Although NAC is often used in the clinic to restore hepatocellular GSH levels to protect against APAP hepatotoxicity, in the context of mitochondrial cholesterol accumulation, which impairs the transport of cytosol GSH into mitochondria, the use of NAC may be inefficient to specifically replenish mGSH stores, requiring the use of permeable GSH prodrugs such as GSH ethyl ester (GSHEE)

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Physiological and Molecular Aspects of Cobalamin Transport. Sub-Cellular Biochemistry, vol