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Oxeiptosis is mediated by KEAP1, phosphoglycerate mutase family member 5, and apoptosis-inducing factor mitochondrion-associated 1 under oxidative stress [6]

Graf and Kastins review catalogues a strikingly broad reported activity profile effects on stress tolerance, pain, thermoregulation, endocrine secretion, and withdrawal states while concluding that the physiological functions and mechanism remain to be established. [2] Schneider-Helmert and Schoenenberger similarly reported that DSIP raised alertness and performance during the waking state and appeared to modulate stress tolerance and coping

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Glutathione facilitates their elimination by binding with these toxins, preventing them from harming vital organs

The findings supported the GSEA results and identified specific affected respiratory states (Fig