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Consequently, it is possible that insulin resistance and hyperinsulinemia may cause endothelial dysfunction, thereby contributing to the pathogenesis of preeclampsia [53]

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Specifically, GLP-1 and GIP are cleaved into two biologically inactive forms, GLP-1 937 (or GLP-1 936 ) and GIP 342

Whether GLP-1 receptor agonists also exert direct effects on immune cells is an area of ongoing preclinical and early clinical research, but this has not been established as a confirmed mechanism in NICE-approved treatment pathways and should be regarded as preliminary