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Treatment should start urgently and should not be delayed for laboratory confirmation if neurological features strongly suggest B12 deficiency

The regulator has requested additional pharmacokinetic data

One of the most profound therapeutic mechanisms of methylcobalamin is its ability to directly support and repair the nervous system

The DAC-modified version is designed for prolonged receptor engagement, while the No DAC form has a shorter activity window that supports pulsatile signaling studies

Clinicians now have more flexibility in tailoring therapy to individual preferences and clinical needs