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Specifically, we observed a stacked, filament-like architecture in which only WD40 domains of COP1 in the top layer are accessible, and a substrate-accessible dimeric architecture that is favored upon co-expression of c-Jun or ETS2

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Thymosin Beta-4 was characterized in a foundational 2005 review in Trends in Molecular Medicine as a regulatory protein involved in cell migration and tissue repair signaling

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