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It is postulated that glutathione inhibits tyrosinase through three primary mechanisms: direct antioxidant activity, chelation of the copper site within tyrosinase via its thiol group, and disruption of tyrosinase's cellular transport to pre-melanosomes

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Therefore, NAC treatment could modify the oxidant/antioxidant imbalance in the lungs, potentially reducing respiratory symptoms, exacerbations, and the deterioration of lung function in COPD patients

Amino acid-dependent control of mTORC1 signaling: A variety of regulatory modes

The study showed a huge variation in liver enzyme levels (image below), but it didnt identify any specific SNP