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Moreover, treating mice with indomethacin, a non-selective cyclooxygenase inhibitor or EP4 antagonist, increased Th17 cell differentiation and enhanced the production of antimicrobial peptides, thereby reducing the bacterial load in the gut and improving host defense against C

By maximizing the output of one component of a system, we inevitably hasten the demise of ecosystems

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The tumor microenvironment at a glance

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