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These components are considered immunological distractors with pro-inflammatory activity, capable of causing dysbiosis and increased intestinal permeability (the so-called leaky gut syndrome), so favoring the development of autoimmune diseases [142]

In mammals, heterodimer formation between the GCLC and GCLM (modifier) subunits results in the GCL holoenzyme complex which has increased catalytic efficiency over the GCLC subunit alone [74]

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