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[70] found in vivo and in vitro studies that GLP-1 and its analog exendin-4 were able to induce an increase in ABCA1 mRNA, increase the expression of apo AI mRNA and promoter, thereby increasing apo AI secretion, indicating that GLP-1 in hepatocytes may mediate the outflow of intracellular cholesterol to apo AI through the ABCA1 pathway

In two-week-old adults of Ap

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Upon stimulation by mitochondrial DNA (mtDNA), lipopolysaccharide-like signals, or peripheral inflammatory cytokines, microglia can adopt a pro-inflammatory activated phenotype for prolonged periods in the central nervous system

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