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Regulation of ferroptosis can be achieved generally by interfering with iron metabolism and ROS metabolism, and the ferroptosis process can be suppressed by iron chelators, lipophilic antioxidants, lipid peroxidation, and the depletion of polyunsaturated fatty acids and correlates with the accumulation of lipid-peroxidation markers (Stockwell et al., 2017)

Unlike GIP, the potent insulinotropic effect of GLP-1 is predominantly preserved in T2D patients and, thus, has led to the development of GLP-1-based therapies for preserving blood glucose control in individuals with T2D

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References Bouchama, A