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Determining optimal doses for psychiatric indications, balancing efficacy against tolerability and metabolic safety, remains a key priority for future trials [15]

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This mechanism involves the conversion of Phosphatidylinositol-4,5-bisphosphate (PIP2) to Phosphatidylinositol-3,4,5-trisphosphate (PIP3), facilitating Akt activation by membrane translocation and phosphorylation by PIP3-dependent kinases