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Pioszak AA, Hay DL

Though the majority of physicians surveyed had been in practice for more than 10 years, most said they had only started prescribing GLP1RA medications recently

The oxidative stress that occurs in the melanocyte, as mentioned before, is possibly the autoimmunity trigger in vitiligo.63 The communication between the melanocyte and the innate immune system seems to occur through the secretion of exosomes, which contain melanocyte-specific antigens, miRNAs, heat shock proteins, and DAMPs (damage-associated molecular patterns).64 These exosomes deliver autoantigens to dendritic cells, which undergo maturation into antigen-presenting cells.70 The DAMP with the highest evidence of association with vitiligo to date is HSP70 (heat shock protein 70).66 HSP70 is a protein from the intracellular chaperone family whose function is to prevent the incorrect folding of other proteins and their aggregation

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Every time you eat, your body releases this hormone