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Due to their high surface area, charge density, and unique reactivity, nanomaterials can provoke cellular oxidative stress, mitochondrial dysfunction, lysosomal impairment, and activation of pro-inflammatory pathways [475,476,477,478]

Furthermore, liraglutide has been shown to inhibit endoplasmic reticulum stress, as well as related apoptosis and inflammatory reactions are mediated through the cAMP/PKA/CREB pathway in BV2 cells that have been exposed to lipopolysaccharide (LPS) or tunicamycin (TM), a mouse-derived microglial cell line [111]