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the terminal half-life of the fusion proteins was greater than 20 h in rats

FOXO4-DRI and related constructs have been studied as tools to better understand these mechanisms, potentially offering models to examine how FOXO-related signaling interacts with neurodegenerative processes

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Immunofluorescent images of glomeruli in Gpx4 fl/fl mice and Gpx4 fl/wt LysMCre + mice

By disrupting the interaction between FOXO4 and p53, FOXO4-DRI reactivates apoptosis in aged cells, clearing them from tissues and promoting systemic rejuvenation (1, 2)