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In doxorubicin-induced cardiotoxicity, PSG-1 confers cardioprotection by selectively suppressing the mitochondrial intrinsic apoptotic pathwayevidenced by enhanced MnSOD activity, preservation of mitochondrial membrane potential, inhibition of mitochondrial permeability transition pore (mPTP) opening, and attenuated cytochrome c release into the cytosol (Ubaidillah et al., 2016)

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Macrophages exist in two polarization states: M1 (pro-inflammatory) and M2 (anti-inflammatory)

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