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Clinical studies (in both obesity and type 2 diabetes) consistently show that GLP-1 RA treatment leads to less hunger, increased satiety, and spontaneous decreases in daily caloric consumption (Zheng et al., 2024)

Body composition, adipokines, bone mineral density and bone remodeling markers in relation to IGF-1 levels in adults with Prader-Willi syndrome

To test whether cell Barr1 deficiency affected M 3 and GLP-1 receptormediated insulin release in vivo, we injected control and -barr1-KO mice with bethanechol (2 mg/kg i.p.), a muscarinic receptor agonist, or exendin-4 (12 nmol/kg i.p.), a GLP-1 receptor agonist

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Table S1 Searching details of the meta-analysis