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In the same direction, using transgenic mice that express the chimeric mouse/human amyloid precursor protein (Mo/HuAPP695swe) and mutant human presenilin 1 (PS1-dE9) together with the sterol regulatory element-binding transcription factor 2 (SREBF2), we have shown that brain cholesterol enrichment accelerates and worsens AD pathology including neuroinflammation, by enhancing the mitochondrial oxidative stress elicited by A [21]

The relationship between autophagy and mitochondrial function is also an important research direction

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Supplemental GSH significantly inhibited activation of NF-B, resulting in a decreased production of DV2 in HepG2 cells

This dual mechanism of action supports both acute and long-term cellular protection strategies

This simple act sterilizes the entry point