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The incidence of Keshan disease is closely associated with very low dietary intakes of selenium and poor selenium nutritional status

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In contrast, it has also been demonstrated that there is an association of low serum UA with AD-related cerebral hypometabolism, but not with A deposition or tau deposition [224], and may even amplify the effects of A involved in AD [225]

By contrast, no significant differences were observed in response to GEP12 at 5 nmol/kg/day (food intake: mean difference 20.0 kcal/day, 95% CI 41.4 to 1.5 kcal/day, p = 0.067

Yang et al., 2022b)