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In research contexts, L-Glutathione has been investigated for its role in maintaining intracellular redox balance through reversible oxidation-reduction cycling between the reduced form (GSH) and oxidized form (GSSG)
In the heart under pressure overload, intracellular copper accumulation can trigger cardiomyocyte cuproptosis via FDX1 activation, leading to lipoylated protein aggregation and mitochondrial damage, thereby exacerbating cardiac injury [12]
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compared to those with acute heart failure, people with COPD had decreased levels of secretoneurin [225]
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