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Nicotinamide adenine dinucleotide (NAD + ) is the cofactor for both these oxidations, being reduced to NADH

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CFTR-dependent enrichment of adult-type enterocyte signatures in mesenchymal stem cell-derived EV39-treated human preterm intestinal epithelial models EV39 treatment in premature intestinal organoids induced a notable enrichment of BEST4+ enterocytes, which are more abundant in pediatric/adult stages than in fetal stages (Figs

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