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Based both on the oddity of non-metazoans having this isoform and the intimate intracellular interaction between these intracellular protozoan parasites and both their vertebrate and invertebrate metazoan hosts, lateral gene transfer from host to parasite seemed a plausible model for the appearance of this unexpected isoform outside of metazoans

Cisplatin treatment upregulated the expression of P66Shc in vivo and in vitro and promoted its mitochondrial translocation to exacerbate ferroptotic cell death and induce cisplatin-induced AKI [173]

However, no increased risk of perianal or stricturing complications has been observed in CD patients (86, 87)

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