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Lyso-IP: Uncovering pathogenic mechanisms of lysosomal dysfunction

The CON ( n =48) were obtained from the Prevention Research Center (PRC) trial and The Johns Hopkins Schizophrenia Center (JHSZC)

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Glutathione

Since PRDX2 has been shown to be glutathionylated by GSH at its cysteine residues to protect against its hyperperoxidation 41 , it is possible that elevated GSH levels in T315I-Bcr-Abl cells might prevent PRDX2 monomer formation, therefore abrogating ROS-induced replication stress

Innate immune activity is detected prior to seroconversion in children with HLA-conferred type 1 diabetes susceptibility