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therefore, the histological and biomechanical analyses necessary for conclusive evidence of the beneficial effects of fibroblast therapy in upright animals were lacking

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Extensive phenotypic characterisation of a human TDP-43(Q331K) transgenic mouse model of amyotrophic lateral sclerosis (ALS)

Lee M, Cho T, Jantaratnotai N, Wang YT, McGeer E, McGeer PL

Using C2C12 mouse myoblasts, the study confirmed that the MGF E-domain peptide increases myoblast proliferation while inhibiting terminal differentiation and critically, that this effect was maintained in the presence of a neutralising anti-IGF-1R antibody directly establishing that MGF E-peptide signals via an IGF-1R-independent mechanism