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Caspase-1-inhibitor AC-YVAD-CMK inhibits pyroptosis and ameliorates acute kidney injury in a model of sepsis

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CLO (100 mg/kg) improved lung tissue architecture more effectively than the lower dose

ROS, formed as a result of the use of contrast agents, can cause damage to the kidneys in several ways: the ionic contrast agent is hyperosmotic therefore can damage the tissue and lead to the accumulation of infected cells, polimorfonuclear leukocytes, at the injured place and to the release of ROS

The rationale is mechanistic: BPC-157 and TB-500 appear to target complementary aspects of tissue repair