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Dysregulated lipid accumulation, defective fatty acid oxidation, and elevated glycolysis-lactate pathway activity lead to mitochondrial damage, and these processes are linked to myofibroblast metabolic shifts in diabetic kidneys (Kang et al., 2015

55 and Kim et al

Whoa nelly The results were exactly in line with what we expect to see, a near 2% decrease in A1c at the highest doses

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