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The concern stems from its activation of the VEGFR2 pathway and FAK-paxillin signaling, which drive angiogenesis and are dysregulated in 50% of human cancers (ovarian cancer, melanoma, and colon adenocarcinoma)

To reach that, further research is required to establish treatments directed to the maintenance and modulation of the BBB integrity through the inhibition/enhancement of the underlying biochemical processes

7, 2016, doi:10.1371/journal.pone.0159198

That caveat established, the preclinical signal is unusually consistent across independent research groups and across a wide range of injury models, which is why the scientific conversation around BPC-157 continues to accelerate