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While stress response can be activated due to antibiotics that target the cell envelope or indirectly damage it, exposure to toxic levels of metals such as zinc will also lead to stress response activation, either through oxidative damage or displacement of other metals from the cell ( zraP inducing antimicrobials, such as the DNA- targeting fluoroquinolone lomefloxacin, as well as compounds that target replication and protein synthesis ( zraP expression level and the presence of a stressor in the bacterial environment to activate this ESR system

Peptides containing methionine residues are prone to oxidation because methionine readily converts to methionine sulfoxide when exposed to oxygen

Previous research suggests that hypoxia exacerbates inflammatory responses [52]

doi: 10.3390/antiox13121571 26 LeeWCDixonSJ

( c ) Fourier shell correlation (FSC) for the density map corresponding to the prefusion structure is shown

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