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Conspicuously diminished expression of GPX4 and NRF2 in psoriatic lesions thereby results in compromised antioxidant capacity and a heightened propensity for ferroptosis, consequently aggravating the breakdown of the epidermal barrier (190, 191)

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The last term is the activation of CBS by oxidative stress [25, 26], which is represented by the concentration of H 2 O 2

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