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The cysteine modification is expected to trigger a conformational change of the Keap1 homodimer, which could dissociate the weak binding site, the DLG motif (latch), from the Keap1 DC domain, while the strong binding site, the ETGE motif (hinge), remains attached to the other Keap1 DC domain
Cost concerns : Using multiple peptides significantly raises research expenses, straining budgets
Zechmann, 2020)
Downregulation of the p53 signaling pathway promotes cellular proliferation and modulates inflammatory responses 71 , while HDAC7 depletion facilitates cell migration, angiogenesis, and extracellular matrix remodeling, collectively contributing to coordinated tissue repair 72
doi: 10.3390/cancers14071652 230 DoepfnerKTBollerDDe LaurentiisAGuerreiroASMarinovMArcaroA
