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EAAC1 is tightly regulated by molecules, including glutamate transporter-associated protein 318 (GTRAP3-18) and miR-96-5p, offering potential therapeutic targets to optimize neuronal GSH levels and mitigate early neurodegeneration (6)

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We have made a number of changes to existing protocols, most notably, the use of defined salt concentrations (0.175 M NaCl) in the working buffer, the use of an ApoC-II peptide as a potent, readily available and cost-effective LPL activator, and the use of the nAngptl4 peptide as an LPL-specific inhibitor

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