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Nevertheless, the homozygote mutant genotype leads to hyperhomocysteinemia only when plasma folate status is low in healthy subjects and in patients with cardiovascular disease, thus subjects with this MTHFR mutation may be sensitive to low folate intake (Christensen et al, 1997

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This low absorption is why some people see no resultsor, conversely, why they might take very high doses to compensate, which increases the risk of side effects

This is not exactly considered a growth hormone deficiency but the continuing march of time

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