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IGF-1 LR3 binds the alpha subunit of the IGF-1 receptor tyrosine kinase, inducing autophosphorylation of the beta subunit on specific tyrosine residues (Y1131, Y1135, Y1136) that create docking sites for insulin receptor substrate (IRS) proteins and Shc adaptor proteins

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No cases of severe injection-site necrosis, abscess, or infection were documented, and there was no evidence of cumulative worsening with repeated doses over time