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This medication mimics the incretin hormone produced by the intestines, encouraging insulin secretion in response to meals while also inhibiting glucagon release

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doi: 10.1016/S0140-6736(23)01127-3 105 KnopFKArodaVRdo ValeRDHolst-HansenTLaursenPNRosenstockJet al

When the medications reach the brain and bind to receptors in the habenula, they reduce the compulsive pull behind behaviors like overeating often described by patients as food noise. Thats one reason why GLP-1s are now being studied as potential tools for treating or preventing substance use disorders

Researchers could not address potential clustering effects that could happen at the clinical site level because of the nature of the study

[10] [11] While the absolute incidence remains uncertain, these reports have prompted heightened awareness and protocol modifications