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10.1371/journal.pone.0068444 114

When the peptide binds to these receptors, it initiates a signaling cascade that increases the expression of lipolytic enzymes, particularly hormone-sensitive lipase, which breaks down stored triglycerides into free fatty acids and glycerol

GLP-1s (which stands for glucagon-like peptide-1) are a class of medications that were originally developed to manage type 2 diabetes

Because many previous reports demonstrated that hypertension alone induces a shift from fatty acid oxidation toward increased glucose utilization in cardiac muscle, hypertension complicated with HFpEF might have additive effects on energy metabolic failure in heart