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When you eat, GLP-1 is released, prompting the pancreas to produce insulin

[5] This rapid breakdown explains why therapeutic GLP-1 receptor agonists have been chemically modified to resist DPP-4 degradation, thereby extending their duration of action and clinical efficacy in managing diabetes and obesity

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This causes damage to pancreatic tissue and stimulates nerve endings, resulting in intense abdominal pain

Amino acid supplementation increases lean body mass, basal muscle protein synthesis, and insulin-like growth factor-I expression in older women

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