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Description

These findings highlight the complex and context-dependent role of ROS in IPF pathogenesis, where both insufficient and excessive ROS can be detrimental

Am J Clin Oncol

Despite the redundancy accorded to this system by the continued production of JNK2, it appears reasonable to conclude that the JNK1 signaling pathway may be playing a role in T-cell receptor initiated Th cell proliferation, differentiation or apoptosis