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doi: 10.1016/J.ENVINT.2018.07.029 84

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One example in diabetic cohorts treated with MitoQ, the clinical protocol was designed combining DM endpoints (HbA1c and endothelial function) in a more preferable way than isolating redox-specific effects from glucose-lowering biomarkers

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The histopathological hallmark of ammonium toxicity is manifested by the emergence of aberrant astrocytes, known as Alzheimer type II astrocytes