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In a paper Bluestone and colleagues published in the AACR journal Cancer Immunology Research , they note that irAEs are either a consequence of an autoinflammatory response, caused by the activation of innate immunity, or an autoimmune response, indicated by the presence of autoantibodies and antigen-specific memory T-cell responses

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Preclinical work had suggested that IGF-1 could increase clearance of beta-amyloid from the central nervous system, and the age-related decline of the GH/IGF-1 axis had been proposed as a contributor to neurodegeneration

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