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doi:10.1002/lary.23376 Neuhauser HK

OS also reduces tetrahydrobiopterin (BH4), a critical cofactor for eNOS, causing it to produce more ROS instead of NO, thereby amplifying endothelial injury (Hernandez-Navarro et al., 2024)

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This parallels the fatigue reported with GLP-1 agonists and semaglutide-specific tiredness

Extensive evidences have highlighted the interaction between mitochondrial metabolic dysfunction, mtDNA damage, mitochondrial dynamics disorders, mitophagy dysregulation, and oxidative stress, while targeting oxidative stress represents a promising strategy for combating mitochondria diseases