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Experimental studies in glioma models revealed that SIRT6 overexpression suppresses tumor growth by promoting histone deacetylation and cell-cycle arrest, whereas SIRT1 limits androgen biosynthesis through FOXO1 deacetylation, thereby restraining tumor progression ( In CRC, SIRT1 mediates -catenin deacetylation, which drives its nuclear exclusion and attenuates the Wnt signaling pathway (114)

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