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10.1016/j.lfs.2018.12.029 5

In DM, impaired BCAAs catabolism may disrupt pulmonary repair mechanisms, potentially exacerbating post-injury complications (Lynch and Adams, 2014)

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Despite manual corrections and deletions, only a few duplicate cases may exist [53, 54]

Similarly, other studies indicate that enhanced PS exposure may result in a pathological erythrocyte procoagulant phenotype, which may contribute to the development of a hypercoagulable state