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This suggests that the effects of second-generation antipsychotics on memory function may be due in part to increased brain levels of PREG and/or PregS (62, 373)

Citrate and the conversion of carbohydrate into fat

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tuberculosis in mouse lung tissue but also enhances its resistance to ROS and reactive nitrogen species (RNS), thereby promoting ferroptosis in lung tissue, leading to pathological damage and inflammation in the mouse lung ( M